ALS Association (ALSA) (amyotrophic lateral sclerosis)
Center for Neuroscience, University of California, Davis
CSIR – Indian Institute of Toxicology Research
Duke Integrated Toxicology Program – Neurotoxicology
IBRO (International Brain Research Organization)
ICEH – Institute for Children’s Environmental Health
Working with health-affected constituencies, in particular learning and developmental disabilities organizations, to educate their members about possible environmental links to various health problems and to nurture their capacity to advocate for policies that protect children from neurotoxicants.
Iowa Center for Advanced Neurotoxicology (ICAN)
Laboratory for Neurotoxicity Studies,Virginia-Maryland Regional College of Veterinary Medicine
Provides expertise in neuroscience, neurochemistry, neurotoxicology, neuropathology, and clinical neurology for studies done in animal models or in vitro.
Learning Disabilities Association (LDA)
Mossakowski Medical Research Center, Polish Academy of Sciences (IMDiK)
Natural Resources Defense Council (NRDC) – provides information on neurotoxicology and children’s health
NEOMA – Neurology, Environmental and Occupational Medical Associates
Neuromuscular Disease Center, Washington University School of Medicine, St. Louis, MO
This is a great site that lists chemicals that cause toxic neuropathies with detailed descriptions.
Neurotoxicology Research Core, Center for Ecogentics and Environmental Health, University of Washington
Neurotoxicology Research Group of the Institute for Risk Assessment Sciences (IRAS) of Utrecht University
Norfolk State University – Center for Materials Research of Govindarajan Ramesh
Research on biochemical and molecular mechanisms involved in metal toxicity particularly arsenic, lead and manganese.
Oregon Institute of Occupational Health Sciences (OHSU)
University of Michigan – Neurotoxicology Laboratory of Rudy Richardson
Mechanistic neurotoxicology with a focus on serine esterases, particularly Neuropathy Target Esterase (NTE), the enzyme whose inhibition by organophosphorus agents, followed by aging is thought to be the principal component in the etiology of organophosphorus compound-induced delayed neuropathy (OPIDN).